My research laboratory is focused on using genetically modified mice to develop models of inflammatory and fibrotic lung diseases. My laboratory has defined the roles of the glycosaminoglycan hyaluronan and its receptors, CD44, TLR4 and TLR2, in regulating lung inflammation and fibrosis in response to non-infectious lung injury.
- Innate Immunity and Matrix Interactions Regulate Lung Injury and Repair
- Molecular Mechanisms of Lung Fibrogenesis
- Role of Chemokines and Chemokine Receptors in Lung Injury and Repair
- Role of MicroRNA Lung Injury
Meet Our Team
Learn more about the scientists, faculty members, investigators and other healthcare professionals of the Jiang Laboratory, whose dedicated efforts lead to groundbreaking discoveries.
Huan C, Yang T, Liang J, Xie T, Cheng L, Liu N, Kurkciyan A, Monterrosa Mena J, Wang C, Dai H, Noble PW, Jiang D.
Sci Rep. 2015 Oct 7;5:14910.
Xu X, Dai H, Geng J, Wan X, Huang X, Li F, Jiang D, Wang C.
Lab Invest. 2015;95(8):846-859.
Dong Y, Geng Y, Li L, Li X, Yan X, Fang Y, Li X, Dong S, Liu X, Li X, Yang X, Zheng X, Xie T, Liang J, Dai H, Liu X, Yin Z, Noble PW, Jiang D, Ning W.
J Exp Med. 2015;212(2):235-252.